Gastritis

1. Pathophysiology

1.1. Acute

1.1.1. Inflammation

1.1.2. Increased gastric acid production

1.2. Chronic

1.2.1. Older Adults

1.2.2. Type A

1.2.2.1. Fundal

1.2.2.2. Immune fundal

1.2.2.3. Rare

1.2.2.4. Atrophy of stomach

1.2.2.4.1. Reduction of Acid

1.2.3. Type B

1.2.3.1. Nonimmune

1.2.3.2. Antrum of Stomach

1.2.3.3. More Common than type A

1.2.3.4. Acid levels are higher than normal

1.2.3.4.1. Duodenal Ulcers

1.2.3.5. Can lead to Gastric Cancer

2. Clinical Presentation

2.1. Abdominal pain

2.1.1. Vague

2.1.2. Tenderness

2.1.3. Bleeding

2.2. Nausea

2.2.1. Vomiting

2.3. Heart Burn

2.3.1. Pain increases after eating

2.3.2. Pain increases when lying flat

3. Risk Factors

3.1. NSAID use

3.2. Age

3.3. Diet

3.4. Stress

4. Epidemiology

4.1. Medication

4.1.1. NSAIDS

4.2. Infection

4.2.1. H. Pylori

4.3. Getetics

4.4. Autoimmune disorder

5. Diagnosis

5.1. Assessment

5.2. Endoscopy

5.3. Lab tests

5.3.1. Blood draw

5.3.2. Stool antigen level

6. Treatment

6.1. Medication

6.1.1. Proton pump inhibitors

6.1.2. Antacids

6.2. Diet

6.2.1. Bland diet

6.2.2. Avoid Spicy Foods

6.3. Stress reduction

6.4. Surgery

6.4.1. Ulcer repair