Gastritis

1. Pathophysiology

1.1. Gug

1.2. § Acute gastritis: H. Pylori. Alcohol NSAIDs. Metabolic disorders, and histamine can cause inflammation and reduces gastric sensations.

1.3. § Chronic: Gastric mucosa degenerates over time from reduced parietal cella and intrinsic factor, lack of T-Cell tolerance and development.

2. § Clinical Presentation

2.1. Heartburn

2.2. Burning/aching in epigastric area ( can get worse or better with food

2.3. Nausea

2.4. Vomiting

3. § Diagnosis

3.1. History and physical exam

3.2. Endoscopy

3.3. H. Pylori testing

3.4. Biopsy

4. Epidemiology

4.1. H. Pylori diagnosis, Good hygiene, Household size, and cooking habits are all leading risk factors for diseases.

4.2. 5% of cases are autoimmune in origin.

5. Risk Factors

5.1. § H. Pylori infection

5.2. NSAID use

5.3. Alcohol use/toxic ingestion

6. § Treatment

6.1. Mild can self-repair

6.2. Dietary changes

6.3. Antacids

6.4. Quit smoking

6.5. Dietary changes

6.6. Stress reduction

6.7. Avoid NSAIDs

6.8. Avoid alcohol