1. New node
2. New node
3. New node
4. New node
5. New node
6. New node
7. Reduce oncotic pressure
7.1. Hypoproteinemic disease
7.1.1. Nephrotic syndrome
7.1.1.1. Primary glomerular lesion
7.1.1.1.1. Minimal change disease
7.1.1.1.2. focal segmental glomerulosclerosis (FSGS)
7.1.1.1.3. Membranous nephropathy
7.1.1.1.4. membranoproliferative glomerulonephritis
7.1.1.1.5. mesangial proliferative glomerulonephritis
7.1.1.2. Systemic cause
7.1.1.2.1. SLE
7.1.1.2.2. IgAV (HSP)
7.1.1.2.3. Infections
7.1.1.2.4. Diabetes
7.1.2. New node
7.1.3. Liver failure
7.1.4. Malnutrition
7.1.4.1. kwashiorkor
7.1.5. New node
7.1.6. New node
7.1.7. Protein losing enteropathy
7.1.7.1. hypertrophic gastritis (Ménétrier disease)
7.1.7.2. milk protein allergy
7.1.7.3. celiac disease
7.1.7.4. inflammatory bowel disease
7.1.7.5. giardiasis
7.1.7.6. intestinal lymphangiectasia
7.1.7.7. right-sided heart dysfunction (post-Fontan procedure
8. Increased capillary permeability
8.1. Severe burns
8.2. Sepsis
8.3. New node
8.4. New node
9. Increased hydrostatic pressure
9.1. From sodium and water retention
9.1.1. New node
9.1.2. Cardiovascular disease
9.1.2.1. Heart failure
9.1.2.1.1. Low output heart failure
9.1.2.1.2. High output heart failure
9.1.2.2. Arteriovenous fitsula
9.1.3. Renal cause
9.1.3.1. Acute glomerulonephritis
9.1.3.1.1. Primary AGN
9.1.3.1.2. Systemic AGN
9.1.3.1.3. Post streptococcal glomerulonephritis
9.1.3.2. Acute and chronic renal failure
9.1.3.3. Drug
9.1.3.3.1. Vasodilator
9.1.3.3.2. dihydropyridine calcium channel blockers
9.1.3.4. Nephrotic syndrome
9.1.4. Idiopathic disease
9.1.4.1. Familial idiopathic edema
9.1.4.2. Non-familial idiopathic edema
9.1.4.3. Pregnancy
9.2. Venous obsruction
9.2.1. Hepatic cirrhosis
9.2.1.1. Genetic
9.2.1.1.1. Alpha-1 antitrypsin deficiency
9.2.1.1.2. Cystic fibrosis
9.2.1.1.3. Wilson disease
9.2.1.2. Infectious
9.2.1.2.1. Viral hepatitis
9.2.1.3. Structural problem of biliary tree
9.2.1.3.1. Biliary atresis
9.2.1.3.2. Alagille syndrome