1. Compication
1.1. Cardiovascular and cerebrovascular
1.1.1. Stroke
1.2. Diabetic retinopathy
1.2.1. Impaired vision
1.2.2. Blindness
1.3. Diabetic neuropathy
1.3.1. Foot wounds
1.3.2. No heat
1.3.3. Wear shoes
1.3.4. See podiatrist
1.4. Diabetic nephropathy
1.4.1. Damage to kidneys, prolonged evaluated BS
1.5. DKA
1.5.1. Presence of ketone in blood and urine
1.6. HHS type 2
1.6.1. No ketone
1.7. Dawn Phenomenon
1.7.1. Happen between 5-8 am
1.7.2. Body increases BS at waking hr
1.7.3. Hyperglycemia in AM
1.7.4. Treatment: Increase insulin or adjustment insulin time
1.8. Somogyi Effect
1.8.1. Rebound Hyperglycemia: Hypo glycemic at night leads to hyperglycemia at night
1.8.2. Treatment: less insulin in the evening
2. Treatments
2.1. Behavior Modification
2.1.1. Diet
2.1.1.1. Limit Carbs
2.1.1.2. Limits Fats
2.1.1.3. Limits Protiens
2.1.2. Exercise
2.1.2.1. Aerobic
2.1.2.2. Running
2.1.2.3. Walking
2.1.2.4. Swimming
2.2. Medication
2.2.1. Insulin
2.2.1.1. Rapid Acting
2.2.1.2. Short acting
2.2.1.3. Intermediate
2.2.1.4. Long acting
2.2.2. Oral Medication
2.2.2.1. Sulfonylures
2.2.2.2. Meglitinides
2.2.2.3. Biguanides
2.2.2.4. Alpha-glycoside inhibitors
2.2.2.5. Thiazolidinedione
3. Assessment
3.1. Hyperglycemia (If I’m hot & dry I must be on a sugar high)
3.1.1. Polyuria
3.1.2. Polyphasic
3.1.3. Polydipsia
3.2. Hypoglycemia (Cold & Clammy give me some candy)
3.2.1. Shakiness
3.2.2. Mental confusion
3.2.3. Sweating
3.2.4. Palpitations
3.2.5. Headache
3.2.6. Lack of coordination
3.2.7. Blurred vision
3.2.8. Seizures
3.2.9. Coma
4. Diagnosis
4.1. Type1
4.1.1. No Insulin production
4.1.1.1. Beta cells located in the islet of Langerhans don’t work (been destroyed) body dosen’t release insulin
4.2. Type2
4.2.1. Not enough Insulin produce or body effective use of insulin
4.2.1.1. Cells quite responding to insulin,