STATES OF CONSCIOUSNESS

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STATES OF CONSCIOUSNESS Door Mind Map: STATES OF CONSCIOUSNESS

1. DEFINITION

1.1. AWARENESS OF EXTERNAL EVENTS & INTENAL SENSATIONS UNDER A CONDITION OF AROUSAL.

1.2. METACOGNITION

1.2.1. THINKING ABOUT YOUR THOUGHTS

2. LEVELS OF AWARENESS

2.1. HIGHER-LEVEL CONSCIOUSNESS

2.1.1. INDIVIDUALS ACTIVELY FOCUS THEIR EFFORTS ON ATTAINING A GOAL

2.1.1.1. DOING MATH OR SCIENCE PROBLEM

2.2. LOWER-LEVEL CONSCIOUSNESS

2.2.1. REQUIRES LITTLE ATTENTION

2.2.1.1. PUNCHING IN A NUMBER ON A CELL PHONE

2.3. ALTERED-STATES OF CONSCIOUSNESS

2.3.1. CAN PRODUCED BY TRAUMA, DRUGS, FATIGUE, HYNOPSIS

2.3.1.1. FEELING EFFECTS OF TAKEN ALCOHOL

2.4. SUBCONSCIOUS CONSCIOUSNESS

2.4.1. WHEN PEOPLE ARE AWAKE

2.4.1.1. SLEEPING & DREAMING

2.5. NO AWARENESS

2.5.1. FREUD'S BELIEF THAT SOME UNCONSCIOUS THOUGHTS ARE TOO LADEN WITH ANXIETY

2.5.1.1. KNOCKED OUT BY A BLOW OR ANESTHETIZED

3. SLEEP AND DREAMS

3.1. SLEEP

3.1.1. NATURAL STATE OF REST FOR THE BODY AND MIND THAT INVOLVES THE REVERSIBLE LOSS OF CONSCIOUSNESS

3.1.1.1. BIOLOGICAL RHYTHMS

3.1.1.1.1. CIRCADIAN RHYTHMS

3.1.1.2. BIOLOGICAL CLOCKS

3.1.1.2.1. DESYNCHRONIZING

3.1.1.2.2. RESETTING

3.1.1.3. SLEEP DEPRIVATION

3.1.1.3.1. LACK OF SLEEP IS STRESSFUL

3.1.1.3.2. STAGES OF WAKEFULNESS & SLEEP

3.1.1.4. SLEEP AND THE BRAIN

3.1.1.4.1. RETICULAR FORMATION

3.1.1.4.2. NEUROTRANSMITTER INVOLVES IN SLEEP

3.1.1.5. SLEEP AND DISEASE

3.1.1.5.1. STROKE ANS ASTHMA

3.1.1.5.2. OBESITY AND HEART DISEASE

3.1.1.5.3. IMMUNE SYSTEM INFECTION

3.1.1.5.4. MENTAL DISORDERS

3.1.1.5.5. ALZHEIMER DISEASE, STROKE AND CANCER

3.1.1.6. SLEEP DISORDERS

3.1.1.6.1. INSOMNIA ( INABILITY TO SLEEP)

3.1.1.6.2. SLEEPWALKING, SLEEPTALKING AND SLEEP EATING

3.1.1.6.3. NARCOLEPSY

3.1.1.6.4. SLEEP APNEA

3.1.1.6.5. NIGHT TERRORS

3.2. DREAMS

3.2.1. COGNITIVE THEORY

3.2.1.1. DREAMS ARE SUBCONSCIOUS COGNITIVE PROCESSING

3.2.1.2. DREAMS ARE A STATE IN WHICH PERSON CAN SOLVE PROBLEMS & THINK CREATIVELY

3.2.2. ACTIVATION-SYNTHESIS THEORY

3.2.2.1. DREAMS AS RESULT OF SYNTHESIS OF NEURAL SIGNALS

4. PSYCHOACTIVE DRUGS

4.1. SUBSTANCE THAT ACT ON NERVOUS SYSTEM

4.1.1. ALTER CONSCIOSNESS

4.1.2. MODIFY PERCEPTIONS

4.1.3. CHANGE MOODS

4.2. CONTINUED USED CAN LEAD TO TOLERANCE

4.2.1. NEED TO TAKE INCREASING AMOUNTS OF DRUGS TO GET THE SAME EFFECT

4.3. PHYSICAL DEPENDENCE

4.3.1. NEED FOR SPECIFIC DRUGS

4.3.2. WITHOUT DRUGS THE PERSON EXHIBIT WITHDRAWAL SYMPTOMS

4.4. PHYSIOLOGICAL DEPENDENCE

4.4.1. STRONG DESIRE TO REPEAT THE USE OF A PARTICULAR DRUG FOR EMOTIONAL REASONS

4.5. ADDICTION

4.5.1. REFERS TO PHYSICAL OR PHYSIOLOGICAL DEPENDENCE OR BOTH

4.5.2. INCREASE DOPAMINE LEVELS IN BRAIN'S REWARD PATHWAYS

4.5.2.1. VENTRAL TEGMENTAL AREA

4.5.2.2. NUCLEUS ACCUMBENS

4.5.2.3. ACTIVATION OF LIMBIC AND PREFRONTAL AREAS OF BRAIN

4.6. DEPRESSANT

4.6.1. HEROIN

4.6.2. TRANQUILIZERS

4.6.3. BARBITURATES

4.7. STIMULATNS

4.7.1. INCREASE THE CENTRAL NERVOUS SYSTEM'S ACTIVITY

4.7.1.1. CAFFEINE

4.7.1.2. NICOTINE

4.7.1.3. AMPHETAMINES ( UPPERS)

4.7.1.4. COCAINE

4.7.1.5. ECSTASY

4.8. HALLUCINOGENS ( PSYCHEDELIC DRUGS)

4.8.1. DRUGS THAT MODIFY A PERSON'S PERCEPTUAL EXPERIENCES AND PRODUCE VISUAL IMAGES THAT ARE NOT REAL

4.8.2. MARIJUANA

4.8.3. LSD ( LISERGIC & DIETHYLAMIDE)

5. HYPNOSIS

5.1. ALTERED STATE OF CONSCIOUSNESS

5.2. FOUR STEPS

5.2.1. 1. MINIMIZING DISTRACTIONS, MAXIMIZING COMFORT

5.2.2. 2. CONCENTRATION ON SOMETHING SPECIFIC

5.2.3. 3. INFORMATION ABOUT WHAT TO EXPECT IN HYPNOTIC STATE

5.2.4. 4. SUGGESTION OF EVENTS OR FEELINGS ALREADY OCCURING

5.3. HIDDEN OBSERVER

5.4. USES

5.4.1. DAMPEN BRAIN PROCESSES EXPERIMENTALLY

5.4.2. HELPS IN UNDERSTANDING EFFECT OF HYPNOSIS AND BRAIN'S FUNCTIONING

5.4.3. MOST EFFECTIVE WHEN COMBINED WITH PSYCHOTHERAPY

6. MEDITATION

6.1. ATTAINING PEACEFUL STATE OF MIND

6.2. MINDFULNESS MEDITATION

6.2.1. FOR DEPRESSION, PAIN ATTACKS AND ANXIETY

6.2.2. FOR CHRONIC PAIN , STRESS AND PSORIASIS

6.3. MEDITATIVE STATE OF MIND

6.3.1. HYPNOGOGIC REVERIE ( FEELING OF WELLNESS)

6.3.2. INCREASED ACTIVATION IN BASAL GANGLIA & PREFRONTAL CORTEX

6.3.3. DECREASES IN ANTERIOR CINGULATE