Get Started. It's Free
or sign up with your email address
Rocket clouds
test 2 by Mind Map: test 2

1. Functioning

1.1. Major Brian Biases

1.1.1. Self Serving Bias

1.1.2. Cognitive Fluency

1.1.3. Confirmation Bias

1.1.4. Implicit Bias

1.1.5. Sunk Cost Fallacy

2. Neurotransmitters

2.1. Autoreceptors

2.2. Post Synaptic Receptors

2.3. Calcium

2.4. agonist

2.5. antagonist

2.6. precursor

2.7. Classic Neurotransmitters

2.7.1. Acetylcholine (ACh)

2.7.1.1. Excitatory Neuromuscular NT

2.7.1.2. Inhibitory NT

2.7.1.3. Nicotonic Receptors

2.7.1.4. Muscarinic Receptors

2.7.2. Monamines

2.7.2.1. Catecholamines

2.7.2.1.1. Norepinephrine (NE)

2.7.2.1.2. Epinephrine

2.7.2.1.3. Dopamine (DA)

2.7.2.2. Indolamine

2.7.2.2.1. Serotonin (5-HT)

2.7.2.3. Monamines Oxidase Enzyme (MAO)

2.7.2.4. Monamine Oxidase Inhibitor (MAO-I)

2.8. Amino Acid Neurotransmitters

2.8.1. Glutamate

2.8.1.1. primary excitatory NT in CNS

2.8.1.2. NMDA receptor

2.8.2. Gamma-Aminobutyric Acid (GABA)

2.8.2.1. GABAa

2.8.2.2. GABAb

2.8.2.3. Most Common Inhibitory NT in CNS

2.8.3. Gylcine

2.8.3.1. Inhibitory in CNS

2.8.3.2. tetanus

2.9. Peptide Neurotransmitters

2.9.1. Oxytocin

2.9.2. Endorphine

2.9.2.1. opiods

2.10. Antidepressants

2.10.1. Selective Serotonin Reuptake Inhibitor (SSRIs)

2.10.2. Tricyclic Antidepressant (TCA)

2.10.3. Monamine Oxidase Inhibitor (MOA-I)

2.11. Opiates

2.11.1. Receptors For Endogenous Opioids

2.11.2. GABA is Antagonistic for DA

2.12. Alcohol

2.13. Marijuana

2.13.1. Tetrahydra-Cannnabinol (TCH)

2.13.1.1. Limbic System= Memory Loss

2.13.1.2. Cerebral Cortex= Sensory Distortion

2.13.1.3. Cerebellum= Balance

2.13.1.4. Imitates Anandamide

2.14. Caffinee

2.14.1. CNS stimulant

2.14.2. Blocks Adenosine Receptors

2.15. Amphetamines

2.15.1. CNS Stimulant

2.16. Cocaine

2.16.1. CNS Stimulant

2.16.2. Crystal Structure

3. Learning and Memory

3.1. Brain Damage

3.1.1. Alzheimer's Disease

3.1.1.1. neurofibularry tangles

3.1.1.2. amyloid beta proteins

3.1.1.3. Damage to Hippocampal Formation

3.1.2. Korsakoff's Syndrome

3.1.2.1. Thiamine Deficiency

3.1.2.2. Alcoholics

3.1.2.3. Damage to Mediodorsal Thalamus

3.1.2.4. Mammillary Bodies Damage

3.2. Cellular Basis

3.2.1. Cellular Modification

3.2.1.1. Neurons Change Shape

3.2.1.2. Calcium Release

3.2.2. Nucleotide Rearrangement

3.2.2.1. DNA/RNA Changes Blocked

3.2.3. Long Term Potentiation

3.2.3.1. NMDA Receptors Activated by Glutimate

3.2.3.2. Receptors Blocked By Magnesium

3.2.3.3. Calcium Pushes out Magnesium

3.3. Classical Conditioning

3.3.1. Eye-Blink Conditioning

3.4. Operant Conditioning

3.4.1. Visual-Spatial Learning

3.4.2. Radial Maze Task

3.4.2.1. 8 of 8 Paradigm

3.4.2.2. Scopolamine

3.4.2.3. Working Memory Paradigm

3.4.2.4. 4 of 8 Paradigm

3.5. Short Term Memory

3.5.1. Working Memory

3.5.1.1. Delayed Response Task

3.5.1.2. Prefrontal Cortex

3.6. Long Term Memory

3.6.1. Implicit Memory

3.6.1.1. Priming

3.6.2. Explicit Memory

3.6.3. Declarative Memory

3.6.4. Procedural Memory

3.7. Memory Consolidation

3.8. Flash Bulb Memories

3.9. Emotional Memories

3.10. Amygdala

3.11. Hippocampus

4. Hippocampus and Memory

4.1. Amnesia

4.1.1. Anterograde Amnesia

4.1.2. Retrograde Amnesia

4.2. Hippoampal Formation

4.2.1. Entorhinal Cortex

4.2.2. Fornix

4.2.3. Perforant Pathway

4.2.4. Dentate Gyrus

4.2.5. Mossy Fibers

4.2.6. CA3

4.2.7. Schaffer Collateral Pathway

4.2.8. CA1

4.3. Damage to Hippocampus

4.3.1. HM (1953)

5. Sleep

5.1. Methods of Studying

5.1.1. Polygraphs

5.1.1.1. EEG

5.1.1.2. EMG

5.1.1.3. EOG

5.2. Epoch